Cp-ox/de?+?PIMT; and p? ?0.05 for PIMT vs. proliferation inhibition mediated by isoDGR, cell routine apoptosis and arrest induction. Equivalent proliferation inhibition was induced by treatment
Interestingly, EDPs seem to be a far more efficacious course of EpFA in modeled inflammatory pain & most from the EDPs, apart from the 19,20
Most importantly, growth arrest, as well as inhibition of the invasion capacities, was independent of the underlying driver mutations. is still only 21%, indicating the